Difference between revisions of Frequently Asked Questions/Myopia/Quotes
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Frequently Asked Questions/Myopia/Quotes (view source)
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==Axial elongation== | ==Axial elongation== | ||
{{quote|“The homeostatic control of eye growth functions to keep images sharply focused on the retina. Therefore, if the eye length increases more slowly than does the focal length, the focal plane will be behind the retina, creating hyperopic defocus on the retina. The same occurs if one puts a negative lens over the eye (Figure 2A). To regain sharp focus, the retina needs to be displaced backward to where the image is. This is done in two ways: the eye is lengthened by increasing the rate of growth or of remodeling of the sclera at the posterior pole of the eye | {{quote|“The homeostatic control of eye growth functions to keep images sharply focused on the retina. Therefore, if the eye length increases more slowly than does the focal length, the focal plane will be behind the retina, creating hyperopic defocus on the retina. The same occurs if one puts a negative lens over the eye (Figure 2A). To regain sharp focus, the retina needs to be displaced backward to where the image is. This is done in two ways: the eye is lengthened by increasing the rate of growth or of remodeling of the sclera at the posterior pole of the eye (Gentle and McBrien 1999 and Nickla et al. 1997), and the retina is pulled back within the eye by the thinning of the choroid, the vascular layer between the retina and sclera (Figure 2B; Wallman et al. 1995 and Wildsoet and Wallman 1995); once distant images are again focused on the retina (emmetropia), both the rate of ocular elongation and the choroid thickness return to normal. | ||
[[File:Ocular_Compensation_for_Lens-Induced_Defocus.jpg|thumb|Ocular Compensation for Lens-Induced Defocus]] | [[File:Ocular_Compensation_for_Lens-Induced_Defocus.jpg|thumb|Ocular Compensation for Lens-Induced Defocus]] | ||
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From https://www.sciencedirect.com/science/article/pii/S0896627304004933 | From https://www.sciencedirect.com/science/article/pii/S0896627304004933 | ||
==Pseudomyopia== | |||
{{quote|“Until the past decade or two, the conventional wisdom had been that myopia was principally genetic in origin both because of the higher incidence of myopia among the children of myopic parents and the large differences in myopia prevalence among ethnic groups (Mutti et al., 2002). This view was weakened by the discovery of homeostatic control of refractive error in animals, including primates. This gave credibility to the epidemiological evidence accumulating over decades that visual factors might contribute to myopia in humans. The evidence is of three types. First, there are epidemiological studies in many countries showing an association between the educational level attained and the prevalence of myopia (e.g., Goldschmidt 1968 and Sperduto et al. 1983), ranging from 3% for unskilled laborers to 30% for those with university educations. Second, a high proportion of young adults who do intensive professional studies (medical, law, engineering, or pilot school) become myopic over the few years of study (e.g., Kinge et al. 2000 and Zadnik and Mutti 1987). Third, cultures in which people lead outdoor lives have little myopia (Morgan and Rose, 2004), but when compulsory education and the other attributes of modern Western culture were introduced to Inuit or American Indian villages, there was a 4-fold increase in the incidence of myopia within one generation (Bear, 1991), although it is difficult to dissociate the visual changes from dietary and other changes (Cordain et al., 2002). The thrust of these findings is that education is associated with an increased prevalence of myopia. The risk factor most discussed as the intervening variable is reading, because the nearness of the page presents the eye with hyperopic defocus. Although the accommodation system reduces this hyperopic defocus, it cannot eliminate it, because accommodation is under negative feedback control, with defocus being the error signal that drives the accommodation output. Therefore, it is plausible that continuous hyperopic defocus during reading drives the emmetropization mechanism to correct this apparent refractive error by making the eye myopic.” | |||
[[File:Near-Work and Myopia.jpg|thumb|Near-Work and Myopia]] | |||
Frequency distribution of refractive errors in four populations of Israeli students. Boys in religious schools, who do much sustained near-work, have a much higher prevalence of myopia than do girls in religious schools or than either girls or boys in secular schools (replotted from Zylbermann et al., 1993.)}} | |||
https://www.sciencedirect.com/science/article/pii/S0896627304004933#BIB235 | |||
{{clear}} | |||
==Hereditary factors of myopia== | |||
{{quote|“In populations with little genetic heterogeneity, such as Inuit populations, studies have revealed that within a generation, the incidence of myopia has risen dramatically in line with the onset of formal education and literacy.3 4 In addition to this evidence for an environmental contribution to the aetiology of myopia, there is also abundant evidence for a genetic influence. These contrasting lines of evidence have stimulated the long running “nature versus nurture” debate, although it is now clear that myopia results from the interaction of environmental and genetic factors.5However, the observed increases in myopia over a generation indicate that the modern myopic epidemic is being fuelled by environmental changes. Furthermore, environmental influences are more easily altered than our genetic make up. Understanding how the visual environment can influence eye growth should therefore be central to any attempts to alter the natural history of myopia.”}} | |||
http://bjo.bmj.com/content/82/3/210.full%C2%A0 | |||
==Infant myopia== | ==Infant myopia== | ||
{{quote|“1. The manifest refractions of 72 children were tracked at regular intervals starting soon after birth and continuing for 9-16 y. Near-retinoscopy, a non-cycloplegic refraction technique, was used for children aged 0-3 y, and non-cycloplegic distance retinoscopy after 3 y. Almost 1400 refractions have been obtained from this group. 2. During the first 6 months the mean spherical equivalent of the group is negative by a small amount. By one year of age the children have an average of 0.5 D of hyperopia which they maintain until 8 y. After 11 y the mean spherical equivalent once again becomes negative, largely because some of the children are becoming myopic. 3. The dispersion of refractions is largest shortly after birth and smallest at 6 y, reflecting the process of emmetropization during the preschool years. 4. The spherical equivalent at 1 y is most predictive of later spherical equivalents. Correlations of spherical equivalent at 1 y with other ages range from 0.43 during the period of emmetropization to 0.76 at some later ages. 5. Children with a negative spherical equivalent in infancy in conjunction with either against-the-rule astigmatism or no astigmatism are more likely to be myopic at school age than children with infantile with-the-rule astigmatism. 6. There is an increased incidence of myopia in children with two (compared to zero or one) myopic parents.”}} | {{quote|“1. The manifest refractions of 72 children were tracked at regular intervals starting soon after birth and continuing for 9-16 y. Near-retinoscopy, a non-cycloplegic refraction technique, was used for children aged 0-3 y, and non-cycloplegic distance retinoscopy after 3 y. Almost 1400 refractions have been obtained from this group. 2. During the first 6 months the mean spherical equivalent of the group is negative by a small amount. By one year of age the children have an average of 0.5 D of hyperopia which they maintain until 8 y. After 11 y the mean spherical equivalent once again becomes negative, largely because some of the children are becoming myopic. 3. The dispersion of refractions is largest shortly after birth and smallest at 6 y, reflecting the process of emmetropization during the preschool years. 4. The spherical equivalent at 1 y is most predictive of later spherical equivalents. Correlations of spherical equivalent at 1 y with other ages range from 0.43 during the period of emmetropization to 0.76 at some later ages. 5. Children with a negative spherical equivalent in infancy in conjunction with either against-the-rule astigmatism or no astigmatism are more likely to be myopic at school age than children with infantile with-the-rule astigmatism. 6. There is an increased incidence of myopia in children with two (compared to zero or one) myopic parents.”}} | ||
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https://www.sciencedirect.com/science/article/pii/S0896627304004933 | https://www.sciencedirect.com/science/article/pii/S0896627304004933 | ||
==Catchy Quotes from [[EndMyopia Discord Server|Discord Chat]]== | |||
* On [[reduction]]: "Oh and 4 weeks is a short time. Even if it was crisp for both eyes you should wait at least 8 weeks. Maybe even 12. When in doubt take the patient route" | |||
* Eating habits impact vision: "for vision improvement , distance vision is the soup and diet is the spice" | |||
* "Not being trapped in four walls 24/7 is the key" | |||